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BPH is the enlargement of the male prostate gland. It can have many associated problems including the need to urinate frequently, difficulty in urinating and inability to completely empty the bladder. The main cause of BPH is age. It’s a very common complaint in older men - indeed over 50% of males 55 years old and over are thought to be suffering with BPH. It’s important for BPH to be properly diagnosed, mainly to assess treatment options and to rule out other more serious problems such as prostate cancer. The difficulty here is many males are embarrassed to seek treatment and indeed some will put up with annoying symptoms for years rather than admit to any problems in this sensitive area! Still others fear being diagnosed with prostate cancer and will spend years worrying about the possibility while being too afraid to know. All this, while quite understandable, coupled with a natural male reluctance to admit to any weakness is counter productive. Why suffer when it’s not necessary? Even if there is a problem, it’s much better to know about it. This gives us back some control and ability to make choices among the many options now available. BPH is what it says - “benign”. Meaning that there is no malignancy. Having been diagnosed then, all worry about cancer should cease! It’s sensible, however, to do everything to prevent prostate cancer - information about which can be found on the site below. Treatments for BPH fall into 3 categories. Drugs, surgery or natural medications. Prostate Surgery - If the symptoms of BPH are severe and causing major problems such as complete inability to empty the bladder, then surgery is the best and probably the only option.. This can take several forms, but the most common is known as TURP. TURP is performed without cutting and access to the prostate is through the urethra. An instrument is inserted which removes any obstructive tissue, easing the pressure thus relieving the BPH symptoms. TURP is a relatively safe operation. Drug Treatments - For those whoses BPH symptoms are not severe - or for those who hate the thought of surgery! - There are two main types of BPH medication. Hormones - For example Finasteride (Proscar). Hormones work by interfering with the male hormone, testosterone. It is a slow acting and long term treatment. The side effects can be impotence and erection failure. Alpha Blockers - These act by relaxing the muscle in the prostate itself which prevents urine flow. This is faster acting than the hormone approach but the side effects are weakness and lethargy. Alternative BPH Treatment These herbal remedies can be extremely effective without the side effects of drugs. For more information visit the site below. This article is for informational use only. It is important to seek medical advice if you are experiencing BPH symptoms. Copyright 2006 Wendy Owen vimax penis enlargement surgery penis elargement tip penile enlargement excercises penis enlarement surgery safe penile enlargment penile enlargment herb pennis enlargement tip free penis enhancement

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If you’re over 40, you’ve got a ticking time bomb in your backside. It’s called benign prostatic hyperplasia…or BPH for short. This is the number one problem in older men. Your prostate is normally about the size and shape of a walnut and is located at the base of your penis. It surrounds your urethra – the tube your urine flows through – and that’s exactly why it’s likely to cause you problems. As you get older, your prostate grows and begins to squeeze the urethra and obstruct normal flow. Most men’s prostates begin enlarging after 40. If you make it to eighty, you have an 80% chance of having BPH. So, what are the symptoms of BPH? • Constant urge to urinate • Frequent nighttime urination • Dribbling or leaking after urination • Difficulty starting urination • A weak stream • Never feeling the bladder is empty Factors that increase your risk The major factors that increase your risk of developing BPH include: • Your medical history • Family history • Diet • Hormone levels Prevention strategies It’s never too early to start preventing BPH…You can save yourself a lot of problems later. And if you already have the symptoms – you can keep them from getting worse. Here are some simple and easy precautions you can take right now: 1. Get enough Omega-3 fatty acids: These are the essential fatty acids in fish, eggs, nuts and flax seed oil. You can get them by eating one of these foods every day…remember most fish that provide Omega-3 are contaminated with mercury…or by taking a supplement. Studies have shown Omega-3 seems to stop the conversion of the chemical that triggers prostate growth. The recommended dose is between 3 and 6 grams a day. 2. Eat healthy: Basically, cut down on the crap … sugars, hydrogenated oils…the things that tend to cause inflammation. Get plenty of protein and veggies. And, to be safe, add a good, natural food base, multivitamin. 3. Watch your DHT levels: Most doctors will tell you that testosterone is the cause of prostate enlargement and give you treatments to reduce it. Now that’s great! The very thing that makes you male and gives you your virility is being taken away from you. Dr. Al Sears wrote, “Testosterone is one of many related steroid hormones. Several are interconverted. Testosterone, for instance, can be converted into estrogens. But testosterone can also be converted into DHT. DHT is 9 times more powerful at stimulating growth of prostate tissue than testosterone is. Testosterone maintains normal health of your prostate but DHT stimulates an overgrowth. DHT sends signals to the prostate tissue, making it swell. As the tissue swells, it impinges on the surrounding urinary and reproductive systems. (DHT is also the chemical that causes men to develop male pattern baldness.) Your body converts testosterone to DHT with an enzyme called 5-alpha reductase. Exposure to stress and steroid related toxins in the environment appear to increase the activity of 5-alpha reductase. This deals a double blow to your manhood. It robs you of testosterone and it increases DHT. But, without the presence of 5-alpha reductase, testosterone will not convert into DHT. And this is the concept behind well-designed BPH treatments. If you can block the action of the 5-alpha reductase, you can prevent and treat prostate enlargement while increasing, not lowering your testosterone. Dr. Sears goes on to say, “You can stop 5-alpha reductase from making DHT with natural supplements. The best inhibitors of 5-alpha reductase come to us in the form of plant sterols. I’ve talked about some of these supplements before. Saw palmetto, pygeum, and pumpkinseed are the “big three”. 4. Get a regular exam: BPH and the worst case, prostate cancer aren’t something to fool around with. Especially if you’re over 40, you should see your doctor for a prostate check-up once a year. Include hormone blood tests, physical exam, and a comprehensive PSA test in your routine. penis enlargment product penis enlagement photo home penile enlargment permanent penis enlargement penis enlargement without pills penis elargement operation compare penis enlagement pills do pennis enlargement pills really work penis enlargment pic

If you’ve ever witnessed someone suffer a stroke, you understand the humbling nature of this disease. It can reduce the mightiest human being to an immobile, helpless creature. Impairment of crucial functions like speech, walking, and control of bowel and bladder can wrench control from the body in a moment. Even perpetually youthful TV personality Dick Clark was struck down by stroke at age 75, despite the outward appearance of perfect health. Clark’s stroke resulted in a six-week hospital stay and, judging from fragmented reports, significant disability. Stroke can be like a devastating fire that strikes without warning, leaving only smoldering rubble. Stroke can so ravage basic bodily functions that often all you can hope for is to regain a portion through rehabilitation. The disease process that underlies stroke requires decades—30 or 40 years—to develop. With that much lead time, why aren’t we better able to detect or stop this crippling disease? The truth is that we are able to predict many, if not most, strokes. Advances in imaging technology allow detection of atherosclerotic plaque that cause stroke years before it becomes a threat. Progress in deciphering the causes of stroke has also leapt forward. Unfortunately, your neighborhood physician still focuses on diagnosing the crisis rather than anticipating it. Physicians prefer to deal with catastrophes and are just not that interested in prevention. Most physicians ask: “Is it time to operate or not?” The medical community obsesses over procedures like carotid endarterectomy (surgical removal of plaque) or carotid stents. Even when a person is afforded the warnings of a “mini-stroke”, or transient ischemic attack (TIA), little more is done once it’s determined that surgery is not necessary—even though this person has high risk for future stroke (50% over 10 years). Let’s flip-flop this approach to stroke. Procedures represent a failure of prevention! Where do strokes come from? Stroke develops when some portion of the brain is deprived of blood. This usually results from a tiny bit of debris that dislodges from an atherosclerotic plaque along the walls of an artery (the same sort that accumulates in coronaries causing heart attack). The sources of debris have been a subject of controversy, but new imaging technologies have settled the question. Any blood vessel that leads from the heart to the brain can be a source. The two carotid arteries on both sides of your neck are a frequent source, as these arteries are prone to develop plaque. (Our discussion will be confined to what are called thromboembolic, or ischemic, strokes, i.e, strokes that occur from plaque that fragments, sending debris to the brain, and will not include the far less common hemorrhagic strokes due to rupture of small vessels in the brain, nor will we discuss atrial fibrillation and other heart causes of stroke. The thromboembolic strokes we discuss cause around 88% of all strokes.) Over the last 10 years, the aorta has been recognized as another important source of stroke. The aorta is the main artery of the body whose branches go to the head, arms, and legs. Atherosclerotic plaque is a live tissue that, through poor diet, inactivity, high cholesterol, overweight, etc., grows and becomes progressively more unstable. At some point, plaque fragments. Little bits break away, traveling to the brain. Fractured plaque also exposes its deeper structures to flowing blood, triggering blood clot formation, which in turn can also fragment and go to the brain. Atherosclerotic plaque is a prerequisite for the most common causes of stroke. If the majority of strokes originate from plaque, why not measure plaque to determine if you’re at risk for stroke? How can we easily, safely, and accurately measure plaque in the carotid arteries and aorta? And if plaque can be measured, can it be shrunk or inactivated to reduce or eliminate risk for stroke? How can plaque be measured? Just 20 years ago, the only practical method of identifying plaque in the carotids or aorta was through angiography, requiring catheters inserted into the body to inject x-ray dye. Angiography was impractical as a screening measure. CT scanning and magnetic resonance imaging (MRI) are emerging as exciting methods of imaging both carotids and aorta. Unfortunately, most centers and physicians are much more focused on the diagnostic uses of these technologies for people who have already suffered stroke or other catastrophe, and application of these devices for preventive uses is still evolving. One exception is when aortic calcification or aortic enlargement is incidentally noted on the increasingly popular CT heart scans; this is an important finding that can signal presence of aortic plaque. The one test that is widely available and can be performed in just about any center is carotid ultrasound. It’s simple, painless, and precise. Two basic observations can be made: 1. Plaque detection—Atherosclerotic plaque can be clearly visualized. If plaque blocks more than 70% of the diameter of the vessel, or if there are “soft” (unstable) elements in plaque, then stroke risk may be high enough to justify surgery or stents. However, if there are plaques that are less severe, substantial risk for stroke may still be present that can be reduced with preventive measures. 2. Carotid intimal-medial thickness—This is a measure of the thickness of the lining of the carotid artery in areas not involved by plaque, but often precedes the development of mature plaque. Carotid intimal-medial thickness also provides an index of body-wide potential for atherosclerotic plaque that can place you at risk for stroke. The aorta, for instance, cannot be well imaged by surface ultrasound but can still be a source for stroke. Increased carotid intimal-medial thickness and carotid plaque are closely associated with likelihood of aortic plaque. The Rotterdam Study of 4000 participants demonstrated that if carotid intimal-medial thickness is greater than normal (1.0 mm), then you can be at risk for stroke (and heart attack), even if no carotid plaques are detected. Carotid ultrasound is the one test you should consider that provides the most information with least effort. Ultrasound is harmless, painless, and can be obtained just about anywhere. Even if your doctor disagrees with your request for a carotid ultrasound, an increasing number of mobile services are popping up nationwide that make this test available for around $100. One important point: many scanners and interpreters will only report whether plaque is present or not. While this is important information, you should request that the carotid-intimal medial thickness be made as well. Not all centers can make this simple measure (because of software requirements), but it doesn’t hurt to try. Any amount of carotid plaque is reason to follow a preventive program, even if the plaque is insufficient to justify surgery. Can plaque be reduced? Can we shrink plaque in carotid arteries and aorta and thereby reduce, perhaps eliminate, these sources of stroke? That question is gaining momentum as effective therapies become available that pack real punch for reducing plaque. Study after study has now documented that plaque can be reduced and, with it, risk for stroke. Reduction in plaque of 10–20% is possible within a year or two. Let’s consider the most potent influences on carotid and aortic plaque growth that need to be considered in a plaque-reducing program. (I assume that you are a non-smoker—if you are a smoker, you first need to concentrate on quitting.) Hypertension Considerable experience documents the power of blood pressure-lowering for prevention of stroke. The most recently updated guidelines, the JNC–VII, recommends a blood pressure of 407 mg/dl heightens stroke risk six-fold. C-reactive protein (CRP) This measure of inflammation is proving to be a useful marker for identifying people at risk for stroke, with increased risk beginning at a level of 0.5 mg/l. High CRP also predicts more rapidly growing carotid plaque. Homocysteine Homocysteine is an important marker of increased likelihood of both carotid and aortic plaque, as well as stroke. In 1997, the European Concerted Action Project reported more than a doubling of stroke when homocysteine levels exceeded 12 mol/l. As homocysteine increases to 20 μmol/l, risk for stroke and heart attack increases an amazing 10-fold over that at a level of 9 μmol/l. Asymmetric dimethylarginine (ADMA) ADMA is recently discovered amino acid whose blood levels can skyrocket up to 10-fold in the presence of hypertension, metabolic syndrome, diabetes, high cholesterol and triglycerides, obesity, and high homocysteine levels. ADMA blocks the action of the amino acid, l-arginine. This mimicry reduces the availability of nitric oxide, a powerful dilator and protector of arteries. ADMA levels in the top 10% predict a six-fold heightened risk for future stroke, and ADMA levels in people with strokes are double that in other people. A carotid ultrasound study in 116 subjects showed that higher blood levels of ADMA are associated with more severe carotid plaque. Because of ADMA’s shared role across a variety of abnormal conditions, correction or blocking the action of ADMA has been suggested as a unique therapeutic tool to reduce stroke risk. Cholesterol Data suggest that lowering cholesterol with statin cholesterol-lowering drugs slows carotid plaque growth and reduce stroke risk approximately 22%. An interesting study from the Cardiovascular Institute at Mt. Sinai School of Medicine in New York using the precise measuring ability of MRI of the carotids and thoracic aorta showed an impressive 20% regression of plaque area with simvastatin (Zocor®) taken for two years. Although guidelines for cholesterol treatment recommend reduction of LDL cholesterol to 100 mg/dl in high-risk persons, a report from the Walter Reed Army Medical Center in Washington, DC, showed that carotid plaque was more effectively reduced when LDL cholesterol of 70 mg/dl or lower was achieved with statin cholesterol drugs. Lower LDL cholesterol may, therefore, be better. Treatment Strategies to Reduce Carotid and Aortic Plaque The essential question: How do we reduce carotid and aortic plaque? If we make this the focus of our efforts, many pieces begin to fall into place. If you’ve had any measure of carotid or aortic plaque such as a carotid ultrasound or aortic calcification on a CT heart scan, you know that you’re at increased risk for stroke. You also have a baseline for future comparison to gauge whether your program is working or not. Because most people have not one but several causes of carotid and aortic plaque, there is no one single treatment that effectively eliminates risk for stroke. Instead, most people require a comprehensive program of healthy diet, exercise, supplements, and medication when indicated. Here, we focus on the nutritional supplements that can be critical components of your plaque-reduction program. Fish oil Fish oil is a cornerstone of your stroke prevention program. Epidemiological observations suggest a strong relationship of fish intake and reduction of stroke risk. Carotid ultrasound studies demonstrate less carotid plaque with greater intakes of fish. A cleverly designed University of Southampton study made the fascinating observation that fish oil transforms the structure of carotid plaque. 150 people with severe carotid plaque scheduled for carotid endarterectomy (surgical removal of the plaque) were given fish oil, sunflower oil, or no treatment over several months while waiting for their procedure. (Delays in the British health system permitted this unique design.) Plaque was removed at surgery and examined. Participants taking fish oil had reduced inflammation in plaque and thicker tissue covering the fatty core, markers of more stable plaque. Those taking sunflower oil or no treatment had unstable plaques with greater inflammation and thinner, less sturdy covering tissue. This suggests that fish oil stabilizes carotid plaque, making it less likely to rupture and fragment. A standard capsule of fish oil (containing 300 mg of EPA + DHA) contains the same amount of omega-3s as a 3 oz serving of cod or halibut; three capsules (900 mg DHA + EPA) contain the equivalent of a serving of farm-raised salmon. The dose that seems to provide greatest protection from stroke, lowers triglycerides (that form abnormal lipoproteins; see above), and reduces fibrinogen, is four capsules per day (1200 mg EPA + DHA). Coenzyme Q10 (CoQ10) Although there are no data specifically addressing whether CoQ10 reduces plaque, it is a marvelously effective way to reduce blood pressure, one of the crucial factors causing carotid and aortic plaque growth. A pooled analysis of eight studies showed that, on average, CoQ10 in daily doses of 50–200 mg reduced systolic blood pressure by 16 mm Hg, diastolic pressure by 10 mm Hg. Data suggest that CoQ10 can reverse abnormal heart muscle thickening (hypertrophy), another manifestation of high blood pressure, strongly suggesting that CoQ10 has benefits beyond just reducing pressure. Supplements to correct the metabolic syndrome Weight loss is, without question, the most immediate and direct path to correction of this dangerous pre-diabetic condition. A drop of even 10–20 lbs yields improvements across the board: increased sensitivity to insulin, increased HDL, and reductions in triglycerides, CRP, fibrinogen, small LDL particles, and blood pressure. Diet and exercise are fundamental components of an effort to lose weight; low carbohydrate or reduced glycemic index diets (e.g., South Beach or Mediterranean) rich in fibers are clearly effective. Several supplements can amplify weight-reduction efforts and be useful adjuncts to your lifestyle program. Among them: White bean extract White bean extract blocks intestinal absorption of carbohydrates by 66%. 1500 mg twice a day with meals yields, on average, 3–7 lbs of weight loss in the first month of use. The only side-effect is excessive gas, due to unabsorbed starches. Glucomannan This unique fiber taken prior to meals absorbs many times its weight in water and thereby fills your stomach. You consequently take in less food. Most people lose around four lbs per month using 1500 mg prior to each meal. Interestingly, glucomannan also blunts the rise in blood sugar after meals, an effect that, by itself, may lead to weight loss. Be sure to take with plenty of water. DHEA This adrenal hormone is key to maintaining physical stamina, mood, muscle mass in men, and libido in women. A recent randomized, placebo-controlled study at Washington University in 56 subjects showed a 13% decline in abdominal fat (fat that drives resistance to insulin) measured by MRI with 50 mg of DHEA per day at bedtime, along with improved sugar control and lower insulin levels. Pectin, beta-glucan Pectin is the soluble fiber in citrus rinds, green vegetables, and apples, also available as a supplement. Beta-glucan is the soluble fiber of oats and is also available as a supplement. Both are wonderful fibers that provide feelings of fullness, lower cholesterol, slow release of sugars, and can yield modest weight reduction. A USC study in 573 subjects using carotid ultrasound showed that greater intake of healthy fibers like pectin and beta-glucan is associated with less carotid plaque growth. Folic acid, vitamins B6 and B12 Dr. Daniel Hackam at the Stroke Prevention and Atherosclerosis Research Centre in Ontario conducted a study using carotid ultrasound in 101 participants treated with folic acid 2.5 mg, vitamin B6 25 mg, and B12 250 mcg per day. Treatment resulted in plaque reduction, especially when homocysteine levels exceeded 14μmol/l at the start, compared to untreated participants who experienced substantial plaque growth. An attempt to clarify the role of homocysteine treatment was made through a National Institute of Health-sponsored study of stroke prevention. 3680 participants with a prior history of stroke were enrolled and given either a “low-dose” (20 mcg folic acid, 0.2 mg B6, 6 mcg B12) or a “high-dose” (2.5 mg folic acid, 25 mg B6, 400 mcg B12) regimen. Although starting homocysteine levels showed a graded association with stroke risk (higher homocysteine levels predicted greater stroke risk), the treatment groups experienced, on average, only a 2 μmol drop in homocysteine levels and no reduction in stroke risk over two years. The study investigators as well as critics have suggested that the study failed due to an insufficient treatment period and that the doses were too low. (The doses we use in our plaque reduction program are folic acid 2.5–5.0 mg, B6 50–100 mg, B12 1000–2500 mcg.) L-arginine L-arginine can be used to overpower the adverse effects of ADMA. L-arginine is emerging as an important carotid plaque-reversing tool. Early reports in animals showed that l-arginine completely halted growth of aortic plaque, and did so more effectively than lovastatin (a cholesterol-lowering drug). In humans, L-arginine reduces blood pressure, abnormal constriction of carotid and coronary arteries, blocks entry of inflammatory cells into plaque, increases sensitivity to insulin, and heightens exercise capacity. Following coronary angioplasty or stent placement, l-arginine results in up to 36% reduction in plaque growth. The average American takes in 5400 mg of l-arginine through food every day. Supplementing with doses of 3000–12,000 mg per day has proven useful to correct many of these phenomena. (We use a dose of 6000 mg of l-arginine powder, twice a day on an empty stomach, dissolved in water, for our plaque regression program.) Does this result in a reduction of stroke risk? The emerging data suggest that l-arginine is likely to exert a powerful plaque-reducing and stroke-preventing benefit, but we await more clinical trial data. Conclusion Reducing stroke risk by reversing carotid and aortic plaque is becoming an everyday reality, with better tools becoming available. To know whether you’re at risk, the best and most available imaging tool is carotid ultrasound, aiming to identify intimal-medial thickness >1.0 mm, or carotid plaque. Any degree of calcification of the aorta, such as on a CT heart scan, is another useful measure of risk. Treatment to reduce risk is multi-faceted but is based on examining all your sources of risk, including metabolic syndrome, small LDL, lipoprotein(a), and C-reactive protein. Fish oil is the one absolutely crucial ingredient in any stroke prevention program. Other supplements can be used in a targeted fashion, depending on the causes identified for your carotid or aortic plaque. Ideally, repeat scanning of your carotids should be done sometime after your program has begun to assess whether you’ve successfully achieved reversal of plaque growth. best penile enlargment pills herbal penis elargement penis enhancement technique real penis elargement compare penile enlargement pills penis enhancement fact penis elargement operation enlagement manhattan penis surgeon penis enlargment pic

Chapter Seven Lilli Ann [Work and Play and Colleen] Many things were starting to happen after the tournament was over. It seemed my life had stopped for a moment in time. I had been working five to six days a week, mostly five. Met a guy, my age called Dan, at the karate studio one evening, he was just watching, and we got talking, and I helped him get a job at Lilli Ann. He was assigned to Mr. Green and would eventually be reassigned down stairs in the packing department. I was assigned in another department, which was one floor lower than his. He started falling in love with a Spanish gal, and wanted me to help him out by asking her why she was so down right rude to him. And so I did, it must have worked because they started dating, thereafter. Well Dan’s brother came into town, he was eighteen-years old, and again Dan and I were both twenty-one. The landlord would not allow two people in their apartment so I talked to my landlady and they ended up renting out the big room. I liked them both, but Dan was a little more level headed. His brother smoked pot night and day, Dan occasionally. About this time my mother said she was coming down to visit me after Christmas, which was not too far off. And so many things were happening. And as the weeks passed by I would often go downtown San Francisco after work and go to the double feature movies, they were older ones but very cheep, .75 cents during the day, before 6:00 PM, and afterwards walk around. I can remember a few times walking down a side street by a little café and Hell’s Angels were hanging out there. One time one of the Hell’s Angels, gave me a strange look but paid me little heed, and went back playing some kind of game. I had to walk around all the motorcycles for they hand them parked in the street, on the sidewalk, and every which way… and them seeing me trying to dodge the bikes to get around them, probably gave them a little groan, one that might have meant, ‘…don’t tip them over sunny.’ And I didn’t bump any. At work a few of the Spanish gals up in Mr. Green’s area were eyeballing me up, but I found out they were married and so I paid little attention to them afterwards. And a few Japanese girls, older women talked to me often, but I never got to dating any of them. Then one evening, after work, Colleen with her sparkling white Catholic seen me waiting for a ride by a street car stand, and asked where I was going, I said down by mission street, and offered to give me a ride. She was around thirty-three years old, whit a healthy looking body, and was hunting I presume—that is, looking for something. Colleen As she drove down Mission Street, she knew exactly where Lilli Ann was, I guess many people did, it was very famous for women’s exclusive clothing, they had dresses in Harpers Bazaar, some famous magazine, and advertised in London, Paris, New York, and here in San Francisco. I closed the window a bit in the car, the air was cool this morning, I told her, but I shouldn’t complain, it was nothing like Minnesota; for weather in December at 57 degrees is like heaven sent; I had heard them say on the radio, that it was going to get to 66 degrees before the end of the day. Not bad, in Minnesota we’d have about forty inches of snow by now, and most likely it would be about three to five below zero. January was the coldest month, in Minnesota usually, reaching many times ten degrees below zero or lower, and February had all the snow it seemed, sometimes twenty inches in one month; sometimes sixteen inches in one day. Some years we had ninety inches of snow. I was inclined to ask her for a date, even though she looked much older than I, but she said first, as I opened the door to get out, “Do you drink wine?” “Occasionally,” I said, for I used to drink some back home, but it was that cheep Ripple crap or Thunderbird, rotten gut stuff. But I didn’t want to tell her that. “The dry wine is even better than the sweet if you have the right bottle, and it’s aged some…” she added as I stood up next to the car, “I’ll pick you up after work, say 4:30 PM, does that sound good?” What could I say, the Cadillac girl was leading, and I had nothing better to do. I hadn’t gone to karate practice going on three weeks now. I think Yamaguchi was a little disappointed in me, surely not his black belt bunch though. “Ok,” I said as I started to turn around and walk inside of the three story building. Things were always happening so fast these days I hardly ever questioned anything. Dan had me meet a friend a week ago, some guy who was selling dope, pot or whatever, we talked and he offered me a job at twice the amount I was making, but I turned him down, I didn’t want to be his or any bodies body guard, end up dead with some heroin stuck in my ass, or down my throat. This was safer, work here at $.1.75 per hour, and just enjoy life; live longer. It was funny, when I stopped to talk to a young man, my age who wanted a quarter, and back in those far off days, they were all over San Francisco, --at any rate, I told him to go get a job, and he asked how much I made in a week, I said $70-dollars, and he laughed, saying: “I make more than that in a day, $75.” Oh well, I guess I still have values. I just couldn’t sit down on the street corner and beg; it wasn’t even a thought. Or should I say, it never occurred to me. ٭ The day went fast, Dan was flirting with his new Spanish girlfriend, who worked in the office at Lilli Ann; I think she was happy I set them up, but I was a little jealous now, I guess I would have like to date her, but I was always drinking, going to movies, and before karate, running around town. No real time I suppose. I think she was wondering why I didn’t smile as much as I did before when I met her halfway going up and down the stairs a few times a day. But I tried. My mother wrote and said he’d be in town now in January. Not too far off. It was 4:35 PM, I just slammed the heavy door behind me to Lilli Ann, and there on the street was that white Catholic, and Dan was not too far behind me, he’s seeing me go to the car, I told myself, not looking in back of me, I’ll hear about it tomorrow. “See yaw later Chick,” Dan said, I think it was to get Colleen’s attention; I turned around and smiled a bit and shook my head. “I did show up, didn’t I, I bet you thought I wouldn’t?” Said Colleen. “Not sure what I thought,” I admitted, and I seem to put on a dumb look. “I always like wine in the fall, --woops, soon to be winter in a week or two.” “Always --” I said-, opening up her car door, and getting in. “Always my new friend, now let’s go to the Bay and look at the Golden Gate.” I nodded my head yes, for I even liked walking along the bank and dock area, by the railroad tracks also. As we got to a certain spot, evening was starting to set in, the once white clouds were turning light-gray, and I opened up the window a little. I loved to grab the moment, absorb what was happening. San Francisco was so very much different than my conservative St. Paul, and it seemed like I was starting to own it a little. There in front of me was the beautiful Golden Gate Bridge, I would never forget it. I had walked across it, seen it a dozen times, and I just never got tired of it; but one thing, I only walked across it once, it is far…longer than one imagines. It was a settling evening. The cars with their horns, the people at work, I was starting to calm down. The night was creeping in. On one hand I was hoping it would never end, and on the other hand, it was a fast pace city for me, it could slow down a bit. “Are you thirsty Chick,” said Colleen. “Oh yes, very much…” I took the bottle from her and drank right out of the top. She pulled out two glasses, then hesitated, and put them back in her back seat saying, “We really don’t need them I see.” I guess I might have seemed a little uncouth, but it was me. For awhile we talked about the earth quake everyone was talking about; how the evangelist’s were saying San Francisco was going to be sunk to the bottom of the ocean. Many people were taking long vacations to get out of town. It was supposed to be on a certain week end coming up (or within the month of January). They talked about it at the bar, at Lilli Ann, every where. She smile, said, “Of course,” as she took a drink. I think she was thinking about her youth; --for whom at twenty-one runs around looking for a glass when you got a bottle. We sat just drinking, and looking at the Bay and the bridge, silent for awhile, some people don’t like too long of a period of silence, but it can be golden, --she lit a cigarette, and so did I, and we took turns drinking. She told a few dirty jokes, and I pretended to think they were funny, and when she laughed I laughed, not because they were funny, but because she was. She commented, “You’ll have to let me know when they have the fabric sales down at Lilli Ann, I want to buy as much as I can.” I didn’t quite understand what she was talking about then, but I did find out later on that they had sales about every four months, and employees could buy fabrics not usable. I would however purchase some for her, during our short time romance. “Let’s go eat,” she commented. “Where…” I said. “I’ll pick up something at a store or restaurant.” “That’s perfect,” I replied, as I put the cork back into the bottle, there was not much left to the wine. Colleen stopped in front of a fancy restaurant, --went inside and ordered some burgers made up for us. “Dolores Street right,” she asked, and I gave her the address, “They’ll taste better relaxing at your apartment.” She said. I explained she was welcome but I only had a small room, and my friend, whom was Dan, she remembered the person who had said, “By Chick”, lived in the other room next to me, --I explained we shared bathrooms. “So she rents out rooms,” she commented. “Yaw, why, you need one?” “Not quite yet, but could be soon, or in a month or so,” she ended her replied with. As she stopped in front of the mansion I lived in, my hunger had changed from food to lust, or so it seemed, the burgers did not seem at all appealing; none the less, we went directly to my room. As we entered the room she looked about, “Quite cute, and yes, you were not kidding, it is small, but cozy, enough for a single man. I had a little dresser by the side of the bed where I kept an ashtray, and a light, along with a little radio. A closet in along side of the bed, a little to the right of the doorway you might say, a window behind me overlooking my bed, and the door to the bathroom on the right also, of the bed; --if I was laying on my back I’d be looking at the doorway in front of me She put the burgers on the small table, took a last drink of the wine, gave it to me, there was one swallow left, I drank it, as she undressed, then she jumped under the covers. She had big breasts and a semi tight body for her age. She was not thin, nor fat, quite healthy looking. I got a hard-on immediately, and like a dog in heat, we pulled our lust together and she grabbed my item and directed it to her warmth. We made love for about 45-minutes, and I fell to my side a bit, rested, and pulled her over to me again, and stuck my penis back into her private area. She was very warm inside, and my body shook as I climaxed. “We should get some sleep Chick,” she said with a chuckle. It seemed she found what she wanted, but I felt a little out classed for some reason. She had a degree I had found out while sitting by the Bay over looking the Golden Gate and she worked as a legal assistant. I couldn’t sleep, so I looked at some of the roof tops of the houses out my window; San Francisco was very complicated for me, all its old and new mixed into a whole, and Colleen laying next to me. But I told myself to go to sleep, tomorrow was another day. As I rolled my body back under the covers, I could not hear anymore car horns, the radio was quiet, Dan and his brother must have fallen asleep, and his girlfriend gone home. The wind was making a bit of noise on the window sill, but that was tranquilizing, if anything. Chapter Eight The Christmas Party Well, Dan was dating the Spanish lady, and Colleen was coming over picking me up on regular bases now. She even got to know the Colonel a little, and Dan and his brother Jack. I think she was eyeing up the little bedroom by Dan’s big room. In-between our dating that is. During this period in San Francisco I was working, and I wasn’t seeing Goesi much, going to the movies as I usually did, and we had a Christmas party coming up in a few days. Mom had written and I expected her to be flying into San Francisco, in two weeks. From here she’d stay a week then fly down to see my brother in Montclair, Southern California. The weather got a little colder also, but why argue it was still in the 50’s during the day, and low 40’s at night. Some rain but not much. I now was running the dogs for the Colonel; I had a hell of a time taking the “Beast,” out. I called him that because he was up to my waste when on all fours, and had teeth almost like a saber tiger; he looked more like a wolf than a dog. He ran like a horse, and I had a choke chain on him; --thank god I could run with him, I think he liked that. And people jumped every which way when they seen us coming: --and a few times he got away from me whereas the panicked started all around me, people jumping far away from the on coming beast, I didn’t blame them. The Confrontation I knew when I left San Francisco, I’d miss the dogs. Matter-of-fact, one night a neighbor came over and was hollering at the Colonel, and threatened her about the dogs, I was in the hallway upstairs listening, had a few beers in me, I came down slowly, and she told the guy to go because I was the one running the dogs, which the guy noticed, and that with my karate, and temper it might not work out too good if he sticks around;” adding, she said, “I think he heard you hollering at me.” “So what, let him come…” and then out of the blue I was five feet from him on the outside stairway, he was two steps down, and the Colonel was against the beam of the door way. “You better take care of them dogs and shut them up before…” “Before I kick you ass, that that…” I leaped toward the man with my hands in the air as to block the man if he thrust the knife at me, and landed on the second to last step, about two feet in front of him, and in a circular motion, threw several blocks to off set his focus, he jumped back, pulled out a two inch knife, he was terrified. “You better not come closer,” he said. I started laughing. “And you mister, better shut your mouth, go home and never, I mean never come around here again, and if I find out you’ve cause any trouble for my landlady, I’ll find you and stick that knife up your ass…….get out of her NOW!!” He moved as fast as he could. Yes, I had my wild moments, as most people have. Said the Colonel, “I hope he doesn’t cause trouble for me, but I sure liked the way you handled him,” and she had a smile half a mile wide. enlagement manhattan penis surgeon vig rx pill natural penis enhancement pills best penile enlargement surgery cheap vigrx pills top penis enargement pills easy enargement free penis surgery way vimax penis enlargement traction device penis enlargment pic

Diabetic frozen shoulder is a major problem. The pain and limited function that it causes can seriously limit the normal activities of day-to-day life. Frozen shoulder is much more common in diabetic patients and this article aims to explore the nature of the Frozen Shoulder – Diabetes connection. There are many ways that diabetes can affect the muscles and joints. Sugar sticks to the collagen in cells and affects its ability to function. Diabetes can damage blood vessels and a poor blood supply results in scarring and damage in the body's elastic tissues. We know that some diabetic patients can have problems with changes in the gristle of their hands - and in men, the penis. Most experts think that diabetic frozen shoulder arises for the same reasons Diabetes is known to affect the shoulder in several ways. Diabetic frozen shoulder seems to be the commonest - with up to 20% of diabetic patients developing frozen shoulder at some time or other. Calcium spots in the tendons and muscle around the shoulder are also seen more commonly in diabetic patients - this probably relates to the fact that high blood sugars can impair blood flow through small vessels. Tendons are particularly vulnerable to this and respond by depositing calcium. These calcium deposits can sometimes be painless but often cause severe discomfort or limited movement. They usually show up on x-rays. Slow healing and impaired nerve function are also common in diabetic patients and contribute to the fact that the frozen shoulder pain takes longer to settle than it does in other, non diabetic, patients. Diabetic patients are much more likely to have problems with their shoulders than others. Insulin dependant diabetics are particularly at risk - with some studies showing that they are six times more likely to develop diabetic frozen shoulder than the rest of the population. We don’t yet really know why diabetic frozen shoulder problems arise but it seems to relate in part to how well each individual controls their blood sugar levels. Textbooks tell you that all shoulder complaints are more common in diabetes but in my experience diabetic frozen shoulder is the most troublesome and most frequent. Diabetics not only get frozen shoulder more often than others but it lasts longer and is more painful for them when they do. Some experts think that shoulder problems in diabetics are so common that they should be regarded as a complication of diabetes and not a coincidental event. There has been a lot of research recently into the frozen shoulder – diabetes link but it is still rather unclear why diabetic patients get such problems with their shoulders. It seems to relate to the effect that diabetes and a high blood sugar has on the collagen containing cells in the body. Collagen is a protein that is involved in making ligaments, tendons and - of course - joint capsules. Diabetic frozen shoulder eventually resolves itself in most cases but can cause a major problem with day to day function for those unlucky enough to suffer from it.